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The Capital Region of Denmark - a part of Copenhagen University Hospital
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The role of ficolins and MASPs in hereditary angioedema due to C1-inhibitor deficiency

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  1. Peripheral memory T cells specific for Arginase-1

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  2. Ficolin-2 reveals different analytical and biological properties dependent on different sample handling procedures

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  1. Immune regulation by fibroblasts in tissue injury depends on uPARAP-mediated uptake of collectins

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  2. Complement Nomenclature-Deconvoluted

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  3. Evasion of Classical Complement Pathway Activation on Plasmodium falciparum-Infected Erythrocytes Opsonized by PfEMP1-Specific IgG

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Hereditary angioedema due to C1-inhibitor deficiency (HAE-C1-INH) causes disturbances in the complement system. However, the influence of HAE-C1-INH on the lectin pathway of complement is unresolved. Thus, we studied the main initiator molecules, enzymes and regulators in the lectin pathway in patients with HAE-C1-INH.
Original languageEnglish
JournalCellular & Molecular Immunology
Volume54
Issue number3-4
Pages (from-to)271-7
Number of pages7
ISSN1672-7681
DOIs
Publication statusPublished - Jul 2013

    Research areas

  • Adaptor Proteins, Signal Transducing, Adult, Aged, Apoptosis Regulatory Proteins, Case-Control Studies, Complement C1 Inhibitor Protein, Complement Pathway, Mannose-Binding Lectin, Female, Glycoproteins, Hereditary Angioedema Types I and II, Humans, Lectins, Male, Mannose-Binding Protein-Associated Serine Proteases, Middle Aged, Young Adult

ID: 39017276