Abstract
YKL-40 is secreted by macrophages, including those in atherosclerotic plaques, neutrophils, and vascular smooth muscle cells. Circulating YKL-40 is elevated in patients with inflammation and increased tissue remodeling. The aim was to examine the sequential changes in serum YKL-40 in patients with acute myocardial infarction (AMI), with and without thrombolytic therapy, as compared with patients with stable coronary artery disease (CAD).
| Original language | English |
|---|---|
| Journal | Coronary Artery Disease |
| Volume | 19 |
| Issue number | 4 |
| Pages (from-to) | 257-63 |
| Number of pages | 7 |
| ISSN | 0954-6928 |
| DOIs | |
| Publication status | Published - 2008 |
Keywords
- Adipokines
- Adult
- Aged
- Biological Markers
- Case-Control Studies
- Female
- Fibrinolytic Agents
- Glycoproteins
- Humans
- Lectins
- Male
- Middle Aged
- Myocardial Infarction
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