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Secondhand cigarette smoke exposure causes upregulation of cerebrovascular 5-HT(1) (B) receptors via the Raf/ERK/MAPK pathway in rats

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  1. Chromogranin A in cardiovascular endocrinology

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  2. Did you know-why does maximal oxygen uptake increase in humans following endurance exercise training?

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  1. Hormonal influences in migraine - interactions of oestrogen, oxytocin and CGRP

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  2. CGRP and migraine; from bench to bedside

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  3. Pre-Chiasmatic, Single Injection of Autologous Blood to Induce Experimental Subarachnoid Hemorrhage in a Rat Model

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Cigarette smoke exposure increases the risk of stroke. Upregulation of 5-hydroxytryptamine 1B (5-HT(1) (B) ) receptors is associated with the pathogenesis of cerebral ischaemia. This study examined the hypothesis that the expression of 5-HT(1) (B) receptors is altered in brain vessels after secondhand smoke (SHS) exposure.
Original languageEnglish
JournalActa physiologica (Oxford, England)
Volume207
Issue number1
Pages (from-to)183-93
Number of pages11
ISSN1748-1708
DOIs
Publication statusPublished - Jan 2013

    Research areas

  • Animals, Cerebrovascular Circulation, Extracellular Signal-Regulated MAP Kinases, Gene Expression, Indoles, MAP Kinase Kinase Kinases, MAP Kinase Signaling System, Male, Phenols, Phosphorylation, Rats, Rats, Sprague-Dawley, Receptor, Serotonin, 5-HT1B, Serotonin, Serotonin Receptor Agonists, Tobacco Smoke Pollution, Up-Regulation, Vasoconstriction

ID: 43093554