Gene expression and 18FDG uptake in atherosclerotic carotid plaques

Sune Folke Pedersen; Martin Græbe; Anne Mette Fisker Hag; Liselotte Højgaard; Henrik Hegaard Sillesen; Andreas Kjær

doi:10.1097/MNM.0b013e32833767e0

Gene expression and 18FDG uptake in atherosclerotic carotid plaques

Sune Folke Pedersen, Martin Græbe, Anne Mette Fisker Hag, Liselotte Højgaard, Henrik Hegaard Sillesen, Andreas Kjær

Department of Clinical Physiology and Nuclear Medicine

98 Citations (Scopus)

Abstract

Metabolic assessment of vascular inflammation by 2-[F]fluoro-2-deoxy-D-glucose positron emission tomography (FDG)-PET is a promising new approach for the evaluation of the vulnerability of atherosclerotic plaques. Quantitative real-time PCR allows measurement of gene expression of markers of atherosclerotic plaque vulnerability. These techniques were applied in advanced atherosclerotic disease to relate metabolism and inflammatory activity to the gene expression profile of the vulnerable atherosclerotic plaque.

Original language	English
Journal	Nuclear Medicine Communications
Volume	31
Issue number	5
Pages (from-to)	423-9
Number of pages	7
ISSN	0143-3636
DOIs	https://doi.org/10.1097/MNM.0b013e32833767e0
Publication status	Published - 1 May 2010

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10.1097/MNM.0b013e32833767e0

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AU - Hag, Anne Mette Fisker

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AU - Sillesen, Henrik Hegaard

AU - Kjær, Andreas

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N2 - Metabolic assessment of vascular inflammation by 2-[F]fluoro-2-deoxy-D-glucose positron emission tomography (FDG)-PET is a promising new approach for the evaluation of the vulnerability of atherosclerotic plaques. Quantitative real-time PCR allows measurement of gene expression of markers of atherosclerotic plaque vulnerability. These techniques were applied in advanced atherosclerotic disease to relate metabolism and inflammatory activity to the gene expression profile of the vulnerable atherosclerotic plaque.

AB - Metabolic assessment of vascular inflammation by 2-[F]fluoro-2-deoxy-D-glucose positron emission tomography (FDG)-PET is a promising new approach for the evaluation of the vulnerability of atherosclerotic plaques. Quantitative real-time PCR allows measurement of gene expression of markers of atherosclerotic plaque vulnerability. These techniques were applied in advanced atherosclerotic disease to relate metabolism and inflammatory activity to the gene expression profile of the vulnerable atherosclerotic plaque.

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Gene expression and 18FDG uptake in atherosclerotic carotid plaques

Abstract

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