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Region Hovedstaden - en del af Københavns Universitetshospital
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The role of inflammation in the pathogenesis of glaucoma

Publikation: Bidrag til tidsskriftTidsskriftartikelForskningpeer review

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  2. Orbital lymphoma

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  3. A review of nasal, paranasal, and skull base tumors invading the orbit

    Publikation: Bidrag til tidsskriftReviewForskningpeer review

  4. Lymphoma of the eyelid

    Publikation: Bidrag til tidsskriftTidsskriftartikelForskningpeer review

  5. The association between multiple sclerosis and uveitis

    Publikation: Bidrag til tidsskriftTidsskriftartikelForskningpeer review

  1. Melanopsin-mediated pupillary light reflex and sleep quality in patients with normal tension glaucoma

    Publikation: Bidrag til tidsskriftTidsskriftartikelForskningpeer review

  2. Lactate-Mediated Protection of Retinal Ganglion Cells

    Publikation: Bidrag til tidsskriftTidsskriftartikelForskningpeer review

  3. Dual Properties of Lactate in Müller Cells: The Effect of GPR81 Activation

    Publikation: Bidrag til tidsskriftTidsskriftartikelForskningpeer review

  4. Clinical and molecular markers in retinal detachment-From hyperreflective points to stem cells and inflammation

    Publikation: Bidrag til tidsskriftTidsskriftartikelForskningpeer review

  5. Conjunctival Goblet Cells, the Overlooked Cells in Glaucoma Treatment

    Publikation: Bidrag til tidsskriftTidsskriftartikelForskningpeer review

Vis graf over relationer
Glaucoma is an ocular disorder characterized by the progressive loss of retinal ganglion cells (RGC) and their axons. There are various hypotheses concerning the cause of RGC death. Previously, glaucoma was defined by high intraocular pressure (IOP); during the past decade, however, glaucoma specialists have acknowledged that elevated IOP is the most important risk factor for glaucoma, but does not define the disease. Other factors such as genetics, blood flow, and excitotoxicity are suggested as potential causal factors for progressive RGC death observed in glaucoma. We review recent studies elucidating a possible role of low-grade inflammation as a causal factor in the pathogenesis of glaucoma.
OriginalsprogEngelsk
TidsskriftSurvey of Ophthalmology
Vol/bind58
Udgave nummer4
Sider (fra-til)311-20
Antal sider10
ISSN0039-6257
DOI
StatusUdgivet - 2013

ID: 42187286