Spironolactone inhibits production of proinflammatory cytokines by human mononuclear cells

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Abstract

The mineralocorticoid receptor antagonist spironolactone (SPIR) reduces the mortality and morbidity in patients with congestive heart failure (CHF). Overexpression of proinflammatory cytokines contribute to the development and progression of CHF.
OriginalsprogEngelsk
TidsskriftImmunology Letters
Vol/bind91
Udgave nummer2-3
Sider (fra-til)87-91
Antal sider5
ISSN0165-2478
DOI
StatusUdgivet - 15 feb. 2004

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