S-adenosyl-L-methionine for alcoholic liver diseases

A Rambaldi; C Gluud

doi:10.1002/14651858.CD002235.pub2

S-adenosyl-L-methionine for alcoholic liver diseases

A Rambaldi, C Gluud

Copenhagen Trial Unit Rigshospitalet

57 Citationer (Scopus)

Abstract

Alcohol is a major cause of liver disease and disrupts methionine and oxidative balances. S-adenosyl-L-methionine (SAMe) acts as a methyl donor for methylation reactions and participates in the synthesis of glutathione, the main cellular antioxidant. Randomised clinical trials have addressed the question whether SAMe may benefit patients with alcoholic liver diseases.

Originalsprog	Engelsk
Tidsskrift	Cochrane Database of Systematic Reviews
Udgave nummer	2
Sider (fra-til)	CD002235
ISSN	1469-493X
DOI	https://doi.org/10.1002/14651858.CD002235.pub2
Status	Udgivet - 2006

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10.1002/14651858.CD002235.pub2

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abstract = "Alcohol is a major cause of liver disease and disrupts methionine and oxidative balances. S-adenosyl-L-methionine (SAMe) acts as a methyl donor for methylation reactions and participates in the synthesis of glutathione, the main cellular antioxidant. Randomised clinical trials have addressed the question whether SAMe may benefit patients with alcoholic liver diseases.",

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AU - Gluud, C

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AB - Alcohol is a major cause of liver disease and disrupts methionine and oxidative balances. S-adenosyl-L-methionine (SAMe) acts as a methyl donor for methylation reactions and participates in the synthesis of glutathione, the main cellular antioxidant. Randomised clinical trials have addressed the question whether SAMe may benefit patients with alcoholic liver diseases.

KW - Humans

KW - Liver Diseases, Alcoholic

KW - Randomized Controlled Trials as Topic

KW - S-Adenosylmethionine

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DO - 10.1002/14651858.CD002235.pub2

M3 - Journal article

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SP - CD002235

JO - Cochrane Database of Systematic Reviews

JF - Cochrane Database of Systematic Reviews

IS - 2

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S-adenosyl-L-methionine for alcoholic liver diseases

Abstract

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