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Normal secretion of the incretin hormones glucose-dependent insulinotropic polypeptide and glucagon-like peptide-1 during gestational diabetes mellitus

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Cypryk, Katarzyna ; Vilsbøll, Tina ; Nadel, Iwona ; Smyczyńska, Joanna ; Holst, Jens Juul ; Lewiński, Andrzej. / Normal secretion of the incretin hormones glucose-dependent insulinotropic polypeptide and glucagon-like peptide-1 during gestational diabetes mellitus. I: Gynecological Endocrinology. 2007 ; Bind 23, Nr. 1. s. 58-62.

Bibtex

@article{3544bf584c2c403a82040bbbd6fe9f7c,
title = "Normal secretion of the incretin hormones glucose-dependent insulinotropic polypeptide and glucagon-like peptide-1 during gestational diabetes mellitus",
abstract = "Gestational diabetes mellitus (GDM) and type 2 diabetes mellitus (DM2) are suggested to be caused by the same metabolic disorder. Defects in gut hormone-dependent regulation of beta-cell function (entero-insular axis) have been proposed to contribute to the pathogenesis of DM2. The aim of study was to evaluate whether an impaired secretion of glucagon-like peptide-1 (GLP-1) and/or glucose-dependent insulinotropic polypeptide (GIP) could play a role in the development of carbohydrate disorders during pregnancy.",
author = "Katarzyna Cypryk and Tina Vilsb{\o}ll and Iwona Nadel and Joanna Smyczyńska and Holst, {Jens Juul} and Andrzej Lewiński",
year = "2007",
month = "1",
day = "1",
language = "English",
volume = "23",
pages = "58--62",
journal = "Gynecological Endocrinology",
issn = "0951-3590",
publisher = "Informa Healthcare",
number = "1",

}

RIS

TY - JOUR

T1 - Normal secretion of the incretin hormones glucose-dependent insulinotropic polypeptide and glucagon-like peptide-1 during gestational diabetes mellitus

AU - Cypryk, Katarzyna

AU - Vilsbøll, Tina

AU - Nadel, Iwona

AU - Smyczyńska, Joanna

AU - Holst, Jens Juul

AU - Lewiński, Andrzej

PY - 2007/1/1

Y1 - 2007/1/1

N2 - Gestational diabetes mellitus (GDM) and type 2 diabetes mellitus (DM2) are suggested to be caused by the same metabolic disorder. Defects in gut hormone-dependent regulation of beta-cell function (entero-insular axis) have been proposed to contribute to the pathogenesis of DM2. The aim of study was to evaluate whether an impaired secretion of glucagon-like peptide-1 (GLP-1) and/or glucose-dependent insulinotropic polypeptide (GIP) could play a role in the development of carbohydrate disorders during pregnancy.

AB - Gestational diabetes mellitus (GDM) and type 2 diabetes mellitus (DM2) are suggested to be caused by the same metabolic disorder. Defects in gut hormone-dependent regulation of beta-cell function (entero-insular axis) have been proposed to contribute to the pathogenesis of DM2. The aim of study was to evaluate whether an impaired secretion of glucagon-like peptide-1 (GLP-1) and/or glucose-dependent insulinotropic polypeptide (GIP) could play a role in the development of carbohydrate disorders during pregnancy.

M3 - Journal article

VL - 23

SP - 58

EP - 62

JO - Gynecological Endocrinology

JF - Gynecological Endocrinology

SN - 0951-3590

IS - 1

ER -

ID: 32267975