Non-synonymous polymorphisms in the FCN1 gene determine ligand-binding ability and serum levels of M-ficolin

Christian Gytz Ammitzbøll, Troels Rønn Kjær, Rudi Nora Steffensen, Kristian Stengaard-Pedersen, Hans Jørgen Nielsen, Steffen Thiel, Martin Bøgsted, Jens Christian Jensenius

26 Citationer (Scopus)

Abstract

The innate immune system encompasses various recognition molecules able to sense both exogenous and endogenous danger signals arising from pathogens or damaged host cells. One such pattern-recognition molecule is M-ficolin, which is capable of activating the complement system through the lectin pathway. The lectin pathway is multifaceted with activities spanning from complement activation to coagulation, autoimmunity, ischemia-reperfusion injury and embryogenesis. Our aim was to explore associations between SNPs in FCN1, encoding M-ficolin and corresponding protein concentrations, and the impact of non-synonymous SNPs on protein function.
OriginalsprogEngelsk
TidsskriftP L o S One
Vol/bind7
Udgave nummer11
Sider (fra-til)e50585
Antal sider10
ISSN1932-6203
DOI
StatusUdgivet - 2012

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