Spring til hovednavigation
Spring til søgning
Spring til hovedindhold
Region Hovedstadens forskningsportal Forside
Hjælp og OSS
Dansk
English
Forside
Hospitaler/afdelinger
Forskere
Publikationer
Aktiviteter
Priser
Søg efter ekspertise, navn eller tilknytning
Natural history and mechanisms of COPD
Peter Lange
*
, Engi Ahmed, Zakaria Mohamed Lahmar, Fernando J Martinez, Arnaud Bourdin
*
Corresponding author af dette arbejde
Lungemedicinsk afd
Medicinske Sygdomme
Oversigt
Fingeraftryk
Fingeraftryk
Dyk ned i forskningsemnerne om 'Natural history and mechanisms of COPD'. Sammen danner de et unikt fingeraftryk.
Sorter
Vægt
Alfabetisk
Keyphrases
Natural History
100%
Lung Function
100%
Inflammatory Changes
66%
Inflammatory Process
66%
Structural Change
66%
FEV1 Decline
66%
COPD Phenotypes
66%
Longitudinal Cohort Study
33%
Smokers
33%
Disease Course
33%
Inflammation
33%
Adult Life
33%
Early Adulthood
33%
Developing Countries
33%
Poor Prognosis
33%
Airway
33%
Enlargement
33%
Neutrophils
33%
Disease Progression
33%
Forced Expiratory Volume in 1 Second (FEV1)
33%
CD8+ T Cells
33%
Tuberculosis
33%
Between-person
33%
T Cell Infiltration
33%
Cell Ratio
33%
New Therapeutic Agents
33%
Asthmatic
33%
Disease Prognosis
33%
Preclinical Stage
33%
Submucosal Glands
33%
Small Airways
33%
Alarmins
33%
Airway Epithelium
33%
Combined Mechanism
33%
Individual Mechanism
33%
Bronchial Biopsy
33%
Promising Areas
33%
Lung Function Trajectory
33%
Ciliated Cells
33%
Type 2 Inflammation
33%
Late Work
33%
Review Mechanism
33%
Structural Lung Disease
33%
Lung Explants
33%
Country Factors
33%
Goblet
33%
Epithelial Metaplasia
33%
Biochemistry, Genetics and Molecular Biology
Cohort Study
100%
Smoking
100%
T Cell
100%
CD8
100%
Cell Infiltration
100%
Metaplasia
100%
Explant
100%
Life Cycle
100%
Immunology and Microbiology
Neutrophil
100%
Cytotoxic T-Cell
100%
Cell Infiltration
100%
Life Cycle
100%
Explant
100%
Medicine and Dentistry
Chronic Obstructive Pulmonary Disease
100%
Disease
45%
Serositis
18%
Smoking
9%
Cohort Analysis
9%
Silo-Filler's Disease
9%
Neutrophil
9%
Therapeutic Agent
9%
Cellular Infiltration
9%
Disease Exacerbation
9%
Cytotoxic T-Cell
9%
Sequela
9%
Life Cycle
9%
Metaplasia
9%
Bronchus Biopsy
9%
Alarmin
9%