Mitochondrial dysfunction in acute and post-acute phases of COVID-19 and risk of non-communicable diseases

Helena Borland Madsen, Jon Ambæk Durhuus, Ove Andersen, Per Thor Straten, Anne Rahbech, Claus Desler

7 Citationer (Scopus)

Abstract

The COVID-19 pandemic, caused by SARS-CoV-2, has resulted in widespread morbidity and mortality, with a significant portion of the affected population experiencing long-term health complications. This review explores the mechanisms of mitochondrial dysfunction in both the acute and post-acute phases of COVID-19, highlighting its impact on various organs and its potential role in the development of non-communicable diseases (NCDs). We discuss how SARS-CoV-2 directly affects mitochondrial function and the role of the virus-induced immune response in exacerbating mitochondrial impairment. This review highlights the critical role of mitochondria in COVID-19 pathogenesis and the importance of addressing mitochondrial health to mitigate acute and chronic effects of the disease.

OriginalsprogEngelsk
Artikelnummer36
Tidsskriftnpj metabolic health and disease..
Vol/bind2
Udgave nummer1
Sider (fra-til)36
ISSN2948-2828
DOI
StatusUdgivet - 4 dec. 2024

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