Intragenic duplication: a novel mutational mechanism in hereditary pancreatitis

Maiken T Joergensen, Andrea Geisz, Klaus Brusgaard, Ove B. Schaffalitzky de Muckadell, Péter Hegyi, Anne-Marie Axø Gerdes, Miklós Sahin-Tóth

19 Citationer (Scopus)

Abstract

In a hereditary pancreatitis family from Denmark, we identified a novel intragenic duplication of 9 nucleotides in exon-2 of the human cationic trypsinogen (PRSS1) gene (c.63_71dup) which at the amino-acid level resulted in the insertion of 3 amino acids within the activation peptide of cationic trypsinogen (p.K23_I24insIDK). The aim of the present study was to characterize the effect of this unique genetic alteration on the function of human cationic trypsinogen.
OriginalsprogEngelsk
TidsskriftPancreas
Vol/bind40
Udgave nummer4
Sider (fra-til)540-6
Antal sider7
ISSN0885-3177
DOI
StatusUdgivet - 2011

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