Interaction between paraoxonase 1 polymorphism and prenatal pesticide exposure on metabolic markers in children using a multiplex approach

Keyphrases

• Multiplex 100%
• Prenatal Pesticide Exposure 100%
• Paraoxonase 1 Polymorphisms 100%
• Metabolic Markers 100%
• Paraoxonase 1 (PON1) 66%
• Leptin 33%
• Genotype 16%
• Homozygote 16%
• Cardiometabolic Risk 16%
• Blood Pressure 16%
• Serum Levels 16%
• Plasminogen Activator inhibitor-1 (PAI-1) 16%
• Exposed Infants 16%
• School-age children 16%
• Body Fat 16%
• Glucagon 16%
• Non-fasting 16%
• Q192R 16%
• Prenatal Environmental Exposure 16%
• Body Fat Content 16%
• Cardiometabolic Disease 16%
• Body Fat Accumulation 16%

Biochemistry, Genetics and Molecular Biology

• Allele 100%
• Paraoxonase 100%
• Aryldialkylphosphatase 100%
• Body Fat 100%
• Leptin 66%
• Blood Pressure 33%
• Genotyping 33%
• Blood Level 33%
• Homozygote 33%
• Lipid Storage 33%
• Glucagon 33%
• Plasminogen Activator Inhibitor-1 33%

Pharmacology, Toxicology and Pharmaceutical Science

• Leptin 100%
• Aryldialkylphosphatase 1 100%
• Biological Marker 50%
• Glucagon 50%
• Plasminogen Activator Inhibitor 1 50%
• Environmental Exposure 50%
• Metabolic Disorder 50%