Gene-environment interaction in atopic diseases: a population-based twin study of early life exposures

Niklas Kahr; Vibeke Naeser; Lone Graff Stensballe; Kirsten Ohm Kyvik; Axel Skytthe; Vibeke Backer; Klaus Bønnelykke; Simon Francis Thomsen

doi:10.1111/crj.12110

Gene-environment interaction in atopic diseases: a population-based twin study of early life exposures

Niklas Kahr, Vibeke Naeser, Lone Graff Stensballe, Kirsten Ohm Kyvik, Axel Skytthe, Vibeke Backer, Klaus Bønnelykke, Simon Francis Thomsen

23 Citationer (Scopus)

Abstract

The development of atopic diseases early in life suggests an important role of perinatal risk factors.

Originalsprog	Engelsk
Tidsskrift	The clinical respiratory journal
Vol/bind	9
Udgave nummer	1
Sider (fra-til)	79-86
ISSN	1752-6981
DOI	https://doi.org/10.1111/crj.12110
Status	Udgivet - 2015

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10.1111/crj.12110

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title = "Gene-environment interaction in atopic diseases: a population-based twin study of early life exposures",

abstract = "The development of atopic diseases early in life suggests an important role of perinatal risk factors.",

author = "Niklas Kahr and Vibeke Naeser and Stensballe, \{Lone Graff\} and Kyvik, \{Kirsten Ohm\} and Axel Skytthe and Vibeke Backer and Klaus B{\o}nnelykke and Thomsen, \{Simon Francis\}",

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T2 - a population-based twin study of early life exposures

AU - Kahr, Niklas

AU - Naeser, Vibeke

AU - Stensballe, Lone Graff

AU - Kyvik, Kirsten Ohm

AU - Skytthe, Axel

AU - Backer, Vibeke

AU - Bønnelykke, Klaus

AU - Thomsen, Simon Francis

PY - 2015

Y1 - 2015

N2 - The development of atopic diseases early in life suggests an important role of perinatal risk factors.

AB - The development of atopic diseases early in life suggests an important role of perinatal risk factors.

UR - https://www.scopus.com/pages/publications/84923229261

U2 - 10.1111/crj.12110

DO - 10.1111/crj.12110

M3 - Journal article

C2 - 24444295

SN - 1752-6981

VL - 9

SP - 79

EP - 86

JO - The clinical respiratory journal

JF - The clinical respiratory journal

IS - 1

ER -

Gene-environment interaction in atopic diseases: a population-based twin study of early life exposures

Abstract

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