Endothelial nitric oxide synthase phosphorylation at Threonine 495 and mitochondrial reactive oxygen species formation in response to a high H₂O₂ concentration

Thomas Jeremy Guterbaum, Thomas Hartig Braunstein, A Fossum, N-H Holstein-Rathlou, C T Torp-Pedersen, Maria Helena Dominguez Vall-Lamora

    17 Citationer (Scopus)

    Abstract

    Hydrogen peroxide (H₂O₂) is produced in vessels during ischemia/reperfusion and during inflammation, both leading to vascular dysfunction. We investigated cellular pathways involved in endothelial nitric oxide synthase (eNOS) phosphorylation at Threonine 495 (Thr(495)) in human umbilical vein endothelial cells (HUVECs) exposed to H₂O₂.
    OriginalsprogEngelsk
    TidsskriftJournal of Vascular Research
    Vol/bind50
    Udgave nummer5
    Sider (fra-til)410-20
    Antal sider11
    ISSN1018-1172
    DOI
    StatusUdgivet - 2013

    Fingeraftryk

    Dyk ned i forskningsemnerne om 'Endothelial nitric oxide synthase phosphorylation at Threonine 495 and mitochondrial reactive oxygen species formation in response to a high H₂O₂ concentration'. Sammen danner de et unikt fingeraftryk.

    Citationsformater