Distinct gene expression signatures in lynch syndrome and familial colorectal cancer type x

Mev  Valentin; Christina Therkildsen; Srinivas Veerla; Mats Jönsson; Inge Bernstein; Ake Borg; Mef Nilbert

doi:10.1371/journal.pone.0071755

Distinct gene expression signatures in lynch syndrome and familial colorectal cancer type x

Mev Valentin, Christina Therkildsen, Srinivas Veerla, Mats Jönsson, Inge Bernstein, Ake Borg, Mef Nilbert

Klinisk Forskningsafdeling AHH

27 Citationer (Scopus)

Abstract

Heredity is estimated to cause at least 20% of colorectal cancer. The hereditary nonpolyposis colorectal cancer subset is divided into Lynch syndrome and familial colorectal cancer type X (FCCTX) based on presence of mismatch repair (MMR) gene defects.

Originalsprog	Engelsk
Tidsskrift	P L o S One
Vol/bind	8
Udgave nummer	8
Sider (fra-til)	e71755
ISSN	1932-6203
DOI	https://doi.org/10.1371/journal.pone.0071755
Status	Udgivet - 12 aug. 2013

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10.1371/journal.pone.0071755

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title = "Distinct gene expression signatures in lynch syndrome and familial colorectal cancer type x",

abstract = "Heredity is estimated to cause at least 20% of colorectal cancer. The hereditary nonpolyposis colorectal cancer subset is divided into Lynch syndrome and familial colorectal cancer type X (FCCTX) based on presence of mismatch repair (MMR) gene defects.",

author = "Mev Valentin and Christina Therkildsen and Srinivas Veerla and Mats J{\"o}nsson and Inge Bernstein and Ake Borg and Mef Nilbert",

year = "2013",

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T1 - Distinct gene expression signatures in lynch syndrome and familial colorectal cancer type x

AU - Valentin, Mev

AU - Therkildsen, Christina

AU - Veerla, Srinivas

AU - Jönsson, Mats

AU - Bernstein, Inge

AU - Borg, Ake

AU - Nilbert, Mef

PY - 2013/8/12

Y1 - 2013/8/12

N2 - Heredity is estimated to cause at least 20% of colorectal cancer. The hereditary nonpolyposis colorectal cancer subset is divided into Lynch syndrome and familial colorectal cancer type X (FCCTX) based on presence of mismatch repair (MMR) gene defects.

AB - Heredity is estimated to cause at least 20% of colorectal cancer. The hereditary nonpolyposis colorectal cancer subset is divided into Lynch syndrome and familial colorectal cancer type X (FCCTX) based on presence of mismatch repair (MMR) gene defects.

U2 - 10.1371/journal.pone.0071755

DO - 10.1371/journal.pone.0071755

M3 - Journal article

C2 - 23951239

SN - 1932-6203

VL - 8

SP - e71755

JO - P L o S One

JF - P L o S One

IS - 8

ER -

Distinct gene expression signatures in lynch syndrome and familial colorectal cancer type x

Abstract

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