Cooperative effect of angiotensin AT(1) and endothelin ET(A) receptor antagonism limits the brain damage after ischemic stroke in rat

Keyphrases

• Ischemic Stroke 100%
• Brain Injury 100%
• Receptor Antagonism 100%
• Angiotensin 100%
• Endothelin-1 (ET-1) 100%
• AT-1 100%
• ETA Receptor 100%
• Cooperative Effects 100%
• Angiotensin II Type 1 Receptor (AT1R) 50%
• Candesartan 50%
• Receptor Antagonist 33%
• Middle Cerebral Artery 33%
• Smooth muscle Cells 33%
• Dual Inhibition 33%
• Neurological Score 33%
• Mean Arterial Pressure 16%
• Combined Treatment 16%
• Ischemic Damage 16%
• Cerebral Arteries 16%
• Acute Treatment 16%
• Wistar Rats 16%
• Endothelin Receptor 16%
• Receptor Expression 16%
• Enhanced Expression 16%
• Cerebral Ischemia 16%
• Occlusion 16%
• Immunohistochemical Study 16%
• Infarct 16%
• Recirculation 16%
• Transient Middle Cerebral Artery Occlusion (tMCAO) 16%
• Neurological Outcome 16%
• Vehicle Control 16%
• Intraluminal Filament 16%
• Experimental Ischemic Stroke 16%
• 2,3,5-triphenyltetrazolium Chloride 16%

Neuroscience

• Eicosanoid Receptor 100%
• Angiotensin 100%
• Brain Injury 100%
• Brain Ischemia 100%
• Endothelin 100%
• Middle Cerebral Artery 50%
• Candesartan 50%
• Receptor Expression 16%
• Endothelin Receptor 16%
• Angiotensin Receptor 16%
• Receptor Antagonist 16%