Central role of glycosylation processes in human genetic susceptibility to SARS-CoV-2 infections with Omicron variants

Frank Geller*, Xiaoping Wu, Vilma Lammi, Erik Abner, Jesse Tyler Valliere, Katerina Nastou, Angus Burns, Morten Rasmussen, Niklas Worm Andersson, DBDS Genomic Consortium, Liam Quinn, Bitten Aagaard, Karina Banasik, Sofie Bliddal, Lasse Boding, Søren Brunak, Nanna Brøns, Jonas Bybjerg-Grauholm, Lea Arregui Nordahl Christoffersen, Maria DidriksenKhoa Manh Dinh, Christian Erikstrup, Ulla Feldt-Rasmussen, Kirsten Grønbæk, Kathrine Agergård Kaspersen, Christina Mikkelsen, Claus Henrik Nielsen, Henriette Svarre Nielsen, Susanne Dam Nielsen, Janna Nissen, Celia Burgos Sequeros, Niels Tommerup, Henrik Ullum, Estonian Biobank Research Team, FinnGen, Lampros Spiliopoulos, Peter Bager, Anders Hviid, Erik Sørensen, Ole Birger Pedersen, Jacqueline M. Lane, Ria Lassaunière, Hanna M. Ollila, Sisse Rye Ostrowski, Bjarke Feenstra

*Corresponding author af dette arbejde

Abstract

The host genetics of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) have previously been studied based on cases from the earlier waves of the pandemic in 2020 and 2021, identifying 51 genomic loci associated with infection and/or severity. SARS-CoV-2 has shown rapid sequence evolution, increasing transmissibility, particularly for Omicron variants, which raises the question of whether this affected the host genetic factors. We performed a genome-wide association study of SARS-CoV-2 infection with Omicron variants, including more than 150,000 cases from four cohorts. We identified 13 genome-wide significant loci, of which only five were previously described as associated with SARS-CoV-2 infection. The strongest signal was a single nucleotide polymorphism in an intron of ST6GAL1, a gene affecting immune development and function, connected to three other associated loci (harboring MUC1, MUC5AC and MUC16) through O-glycan biosynthesis. Our study provides robust evidence for individual genetic variation related to glycosylation, translating into susceptibility to SARS-CoV-2 infections with Omicron variants.

OriginalsprogEngelsk
TidsskriftNature Genetics
Vol/bind58
Udgave nummer2
Sider (fra-til)299–306
Antal sider21
ISSN1061-4036
DOI
StatusUdgivet - feb. 2026

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