APOA5 Deficiency Causes Hypertriglyceridemia by Reducing Amounts of Lipoprotein Lipase in Capillaries

Keyphrases

• Capillary 100%
• Lipoprotein Lipase 100%
• Hypertriglyceridemia 100%
• APOA5 100%
• Angiopoietin-like 33%
• Triglyceride Level 26%
• Plasma Triglycerides 26%
• Wild Mice 13%
• Carboxyl 13%
• Binding Site 6%
• Lipoproteins 6%
• Specific Antibodies 6%
• Lipoprotein Lipase Activity 6%
• Amino Acids 6%
• Recombinant 6%
• Plasma Lipid Levels 6%
• Human Patients 6%
• Cell Culture Experiment 6%
• Antibody Function 6%
• Biochemical Studies 6%
• Ability to Change 6%
• Brown Adipose Tissue 6%
• Terminal Residue 6%
• Terminal Sequence 6%
• Plasma Triglyceride Metabolism 6%

Immunology and Microbiology

• Capillary 100%
• Triacylglycerol Blood Level 100%
• Angiopoietin Related Protein 100%
• Mouse 60%
• Wild Type Mouse 40%
• Carboxy Terminal Sequence 40%
• Surface Property 20%
• Monospecific Antibody 20%
• Cell Culture 20%
• Lipid Blood Level 20%
• Binding Site 20%
• Lipid Level 20%
• Brown Adipose Tissue 20%

Biochemistry, Genetics and Molecular Biology

• Diacylglycerol Lipase 100%
• Hypertriglyceridemia 100%
• APOA5 100%
• Triacylglycerol Blood Level 41%
• Angiopoietin 41%
• Mouse 25%
• Wild Type Mouse 16%
• C-Terminus 16%
• Binding Site 8%
• Monospecific Antibody 8%
• Amino Acids 8%
• Metabolic Pathway 8%
• Apolipoprotein 8%
• Triacylglycerol Lipase 8%
• Pancreatic Lipase 8%
• Blood Lipids 8%
• Cell Culture 8%
• Surface Property 8%
• Lipid Level 8%