A novel KCND3 gain-of-function mutation associated with early-onset of persistent lone atrial fibrillation

Morten Salling Olesen, Lena Refsgaard, Anders Gaarsdal Holst, Anders Peter Larsen, Søren Grubb, Stig Haunsø, Jesper Hastrup Svendsen, Søren Peter Olesen, Nicole Schmitt, Kirstine Calloe

    106 Citationer (Scopus)

    Abstract

    Atrial fibrillation (AF) is the most common cardiac arrhythmia, and early-onset lone AF has been linked to mutations in genes encoding ion channels. Mutations in the pore forming subunit KV4.3 leading to an increase in the transient outward potassium current (Ito) have previously been associated with the Brugada Syndrome. Here we aim to determine if mutations in KV4.3 or in the auxiliary subunit K(+) Channel-Interacting Protein (KChIP) 2 are associated with early-onset lone AF.
    OriginalsprogEngelsk
    TidsskriftCardiovascular Research
    Vol/bind98
    Udgave nummer3
    Sider (fra-til)488-95
    Antal sider8
    ISSN0008-6363
    DOI
    StatusUdgivet - 1 jun. 2013

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